I-Thrombophilia yinkxalabo yombutho we-circulatory system, kwaye ivela ekutshintsheni kwintsholongwane ekubunjweni kunye neepropati zegazi. Esi sifo sikhokelela ngokuthe rhoqo kwi-thromboembolism yeemikhumbi ezinobungozi, i-thrombosis yeendawo ezahlukileyo. Uninzi lwabantu bonke banokugula ngenxa yesifo emva kokuhlinzwa, ngexesha lokukhulelwa, ngenxa yokugqithisa ngokomzimba okanye ukugula. Ukuba ukrokrelwa kwe-tobobylia, kufuneka ufumane ukuhlolwa kwegazi ukusuka kwintlambo kwaye, njengesiqhelo, kwisisu esingenalutho.
Yintoni ebangela i-thrombophilia?
Esi sifo sinokuthi sibe nomlinge ofunyenweyo, obonakaliswa kwiziphene kwiinkqubo zokugawula igazi okanye kwi-pathology yeeseli. Kwaye kunye nesizathu se-thrombophilia sinokungabi naso.
Nangona kunjalo, uhlalutyo lwe-genetic thrombophilia ukuya kwi-50% yamatyala linikeza umphumo omuhle. Oku kubonisa ukuba i-inheritance isingeniso kwi- thrombosis kwizigulane ezininzi. Ukugula okunjalo kungabangelwa zizifo ze-genetic kunye nokuguquka kwegazi kunye ne-antiticoagulation system.
Yintoni uhlalutyo lwe-thrombophilia?
Kuza kube yimhla, eyona nto inolwazi kuhlolwa kwegazi nge-thrombophilia. Ngesi sifo, ukuhlolwa kwegazi kubonisa inani elongeziweyo leeplatelet kunye neeseli ezibomvu zegazi. Umthamo we-erythrocytes ukwanda ngokubhekiselele kumthamo wegazi.
Izinga legazi lichongiwe lizinga lempahla, egalelo ekutshatyalaliswa kwe-thrombi, ebizwa ngokuba yi-D-dimer. Nge-thrombophilia, inani lalo liyakhula.
Ukuvavanywa komsebenzi wokuqhawula igazi kuqhutyelwa ngenxa yokuhlalutya i-APTT (ixesha elisebenzayo le-thromboplastin). Esi sifo sibonakala ngokuhla kwe-APTT.
Ukulungiswa okukhethekileyo kokuhlalutya kwe-genetic thrombophilia akudingekile, isampuli yegazi iyenziwa ngendlela eqhelekileyo kunye nendlela yokuphila yokugula.